Pathogenesis of herpetic stromal keratitis in CCR5- and/or CXCR3-deficient mice.

Essential contribution of CCL3 to alkali-induced corneal neovascularization by regulating vascular endothelial growth factor production by macrophages.

The chemokine receptor CCR5 plays a key role in the early memory CD8+ T cell response to respiratory virus infections.

Expression of the chemokine antagonist vMIP II using a non-viral vector can prolong corneal allograft survival.

C-C chemokine receptor 5 on pulmonary fibrocytes facilitates migration and promotes metastasis via matrix metalloproteinase 9.

Functional Th1 cells are required for surgical adhesion formation in a murine model.

CCR5 is involved in controlling the early stage of Cryptosporidium parvum infection in neonates but is dispensable for parasite elimination.

Chemokine-mediated rapid turnover of myeloid-derived suppressor cells in tumor-bearing mice.

Combined CXCR3/CCR5 blockade attenuates acute and chronic rejection.

Combined inhibition of CCL2, CX3CR1, and CCR5 abrogates Ly6C(hi) and Ly6C(lo) monocytosis and almost abolishes atherosclerosis in hypercholesterolemic mice.

CCR5-dependent regulatory T cell migration mediates fungal survival and severe immunosuppression.

Lack of CCR5 on dendritic cells promotes a proinflammatory environment in submandibular glands of the NOD mouse.

Plasmacytoid dendritic cells induce NK cell-dependent, tumor antigen-specific T cell cross-priming and tumor regression in mice.

A small-molecule compound targeting CCR5 and CXCR3 prevents airway hyperresponsiveness and inflammation.

Pulmonary CXCR2 regulates VCAM-1 and antigen-induced recruitment of mast cell progenitors.

Antibody-mediated rejection of cardiac allografts in CCR5-deficient recipients.

Y-box binding protein-1 controls CC chemokine ligand-5 (CCL5) expression in smooth muscle cells and contributes to neointima formation in atherosclerosis-prone mice.

Cigarette smoke-induced pulmonary inflammation, but not airway remodelling, is attenuated in chemokine receptor 5-deficient mice.

Deletion of CCR2 but not CCR5 or CXCR3 inhibits aortic aneurysm formation.

Dynamic imaging of chemokine-dependent CD8+ T cell help for CD8+ T cell responses.